This study investigates the role of two endogenous inhibitory mechanisms; exercise-induced hypoalgesia (EIH) and a conditioning painful stimulus (CPM) on passive joint range of motion, passive resistive torque and pain sensitivity. The study is a randomized, repeated-measures cross-over study.
The increase in range of motion following stretching is related to changes in the tolerance to stretch. The mechanistic relationships behind these changes in subject sensation are however unknown but could be related to endogenous modulation of pain.
Study Type
INTERVENTIONAL
Allocation
RANDOMIZED
Purpose
BASIC_SCIENCE
Masking
SINGLE
Enrollment
19
An EIH response was induced by a 3-minute isometric contraction of the hand flexor muscles at 25% of maximal voluntary contraction (MVC).
Two bouts of 30 seconds of static stretching (SS) of the knee flexors.
The participants rested in a seated position for 10 minutes
University College of Northern Denmark
Aalborg, Denmark
Knee extension range of motion
Passively induced knee extension range of motion was measured using the Biodex system 4 pro isokinetic dynamometer
Time frame: Passive knee extension range of motion was measured as a baseline just prior to the induction of exercise-induced hypoalgesia (EIH), Static stretch (SS), Rest and a conditioning painful stimulus (CPM)
Knee extension range of motion
Passively induced knee extension range of motion was measured using the Biodex system 4 pro isokinetic dynamometer
Time frame: Passive knee extension range of motion was measured 30 seconds after the induction of EIH, SS, Rest and CPM
Knee extension range of motion
Passively induced knee extension range of motion was measured using the Biodex system 4 pro isokinetic dynamometer
Time frame: Passive knee extension range of motion was measured 10 minutes after the induction of EIH, SS, Rest and CPM
Passive resistive torque
Passive resistive torque during the passive knee extension motion was measured using the Biodex system 4 pro isokinetic dynamometer.
Time frame: Passive resistive torque was measured as a baseline just prior to the induction of EIH, SS, Rest and CPM
Passive resistive torque
Passive resistive torque during the passive knee extension motion was measured using the Biodex system 4 pro isokinetic dynamometer.
Time frame: Passive resistive torque was measured 30 seconds after the induction of EIH, SS, Rest and CPM
Passive resistive torque
Passive resistive torque during the passive knee extension motion was measured using the Biodex system 4 pro isokinetic dynamometer.
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A CPM response was induced by way of the cold pressor test.
Time frame: Passive resistive torque was measured 10 minutes after the induction of EIH, SS, Rest and CPM
Pressure pain thresholds
Pressure pain thresholds was measures using a handheld pressure algometer (Somedic AB, Hörby, Sweden.
Time frame: Pressure pain thresholds were measured as a baseline just prior to the induction of EIH, SS, Rest and CPM
Pressure pain thresholds
Pressure pain thresholds was measures using a handheld pressure algometer (Somedic AB, Hörby, Sweden.
Time frame: Pressure pain thresholds were measured 30 seconds after the induction of EIH, SS, Rest and CPM
Pressure pain thresholds
PRessure pain thresholds was measures using a handheld pressure algometer (Somedic AB, Hörby, Sweden.
Time frame: Pressure pain thresholds were measured 10 minutes after the induction of EIH, SS, Rest and CPM