The COVID-19 pandemic highlights the importance of the prognosis of co-morbidities, such as coronary artery disease, which significantly increase the risk of mortality in patients infected with SARS-CoV2. Investigators have recently studied the complex links between respiratory infections, particularly pneumonia, and type 2 myocardial infarction (MI) in many respects. The etiology of type 2 MI is based on an imbalance of myocardial oxygen supply/need in the absence of rupture/erosion of atheromatous plaques. Based on the RICO survey data, the investigators investigated whether COVID-19-related sepsis and/or respiratory failure could be an underlying mechanism of MI2.
Study Type
OBSERVATIONAL
Enrollment
35
Biological data
Clinical data
Chu Dijon Bourgogne
Dijon, France
Characterizing type 2 myocardial infarction associated with CoV-2 SARS infection
Time frame: Through study completion, an average of 1 year
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