The development of AKI (Acute Kidney Injury) after cardiac arrest is a common factor associated with mortality. In this study, we aimed to evaluate the factors that increase the risk of developing AKI after cardiac arrest. For the study, patients were evaluated for the incidence of acute kidney injury (AKI), factors that increase the risk of AKI, and the impact of AKI on mortality. In this process, demographic data, comorbidities, hemodynamic data, laboratory data, clinical symptoms and findings recorded in the electronic decision support system were recorded and analyzed.
This retrospective study was performed on patients hospitalized in intensive care units after cardiac arrest between 2013 and 2022. Patients who met the inclusion criteria were included in the study. Urea, creatinine values, and whether AKI was diagnosed or not were obtained from the data registry system. Statistical evaluation was performed using univariate binary logistic regression analysis. Demographic data, location of cardiac arrest (in-hospital, out-of-hospital), and duration of CPR were recorded. In addition, the causes of cardiac arrest, HR/MAP (heart rate/average arterial pressure) ratio, and whether VSP (vasopressor) or INO (inotropic) medication was needed were recorded.
Study Type
OBSERVATIONAL
Enrollment
82
Bakırköy Dr. Sadi Konuk Training and Research Hospital
Istanbul, Turkey (Türkiye)
Acute Kidney Injury (AKI)
Creatinine values in the first 48 hours of intensive care unit admission after cardiac arrest
Time frame: Within 48 hours after intensive care unit admission
AKI incidence
Determination of the incidence of AKI development after cardiac arrest
Time frame: Within 48 hours after intensive care unit admission
Mortality
Impact of AKI development on mortality in the early post-cardiac arrest period
Time frame: Clinical and laboratory data within 48 hours after intensive care unit admission will be used
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